Ruminococcus torques are more abundant in the colonic tissue of patients with Crohn’s disease who have upper gastrointestinal tract involvement than those who do not. It may serve as a potential biomarker for predicting this complication.
Ruminococcus torques is also a risk factor for hospitalization and severe COVID-19, according to a Mendelian randomization study. It may affect the host’s immune response and inflammation through its metabolic pathways.
Ruminococcus torques are influenced by the spatial and temporal factors of the gut microbiota, such as the location of the sample, the age of the host, and the season of the year. It shows a higher relative abundance in the ileum than in the colon, in older hosts than in younger ones, and in winter than in summer.
Ruminococcus torques are involved in the pathogenesis of NAFLD, or non-alcoholic fatty liver disease, along with type 2 diabetes, a common comorbidity of diabetes4. It may alter the urine metabolites of the host, such as amino acids, fatty acids, and bile acids, which are related to glucose and lipid metabolism.
Kingdom: Bacteria
Phylum: Firmicutes
Class: Clostridia
Order: Clostridiales
Family: Ruminococcaceae
Genus: Ruminococcus
Species: R. torques
The structure of Ruminococcus torques are:
Ruminococcus torques is a Gram-positive bacterium, which means that it has a thick layer of peptidoglycan in its cell wall that retains the purple stain of the Gram staining method.
Ruminococcus torques is an anaerobic bacterium, which means that it does not require oxygen for its growth and metabolism. It can use alternative electron acceptors, such as nitrate, sulfate, or fumarate, to generate energy.
Ruminococcus torques is a rod-shaped bacterium, which means that it has a cylindrical or elongated morphology. It can form chains or clusters of cells, depending on the environmental conditions.
Ruminococcus torques is a cellulolytic bacterium, which means that it can degrade cellulose, a complex polysaccharide that forms the main component of plant cell walls. It produces enzymes, such as endoglucanases, cellobiohydrolases, and beta-glucosidases, that break down cellulose into glucose and other sugars.
Ruminococcus torques is a symbiont of the gut ecosystem, which means that it lives in association with other organisms, such as humans and other mammals, and provides them with some benefits, such as short-chain fatty acids, vitamins, and amino acids. It also influences the immune system, the intestinal barrier, and the brain function of the host.
The antigenic types of Ruminococcus torques are not well-studied. Still, some studies have suggested that it may have different strains based on the presence or absence of particular genes or metabolic pathways.
For example, a study by Togo et al. found that Ruminococcus torques ATCC 27756 had a gene cluster for the biosynthesis of exopolysaccharides, which may affect its antigenicity and adhesion to the intestinal mucosa. Another study by Ze et al. found that Ruminococcus torques were one of the dominant species involved in the human colon’s breakdown of resistant starch and that it had different metabolic profiles depending on the type of starch substrate.
It has been associated with various diseases, such as inflammatory bowel disease (IBD), irritable bowel syndrome (IBS), and type 2 diabetes mellitus (T2DM) with non-alcoholic fatty liver disease (NAFLD). The pathogenesis of Ruminococcus torques is not fully understood, but some possible mechanisms are:
Ruminococcus torques can degrade mucin, which is a protective layer of the intestinal epithelium, and increase the permeability of the gut barrier. It may allow the translocation of bacterial products and antigens into the bloodstream, triggering inflammation and immune responses.
Ruminococcus torques can produce short-chain fatty acids (SCFAs), such as the nutrients butyrate, propionate, and acetate are necessary for preserving intestinal homeostasis and adjusting host metabolism. However, excessive or imbalanced SCFAs may also have adverse effects, such as altering the gut pH, affecting intestinal motility, and influencing insulin sensitivity.
Ruminococcus torques can interact with other gut microbes and influence their composition and function. For example, Ruminococcus torques may compete with beneficial bacteria, such as Bifidobacterium and Lactobacillus, for nutrients and niches or cooperate with pathogenic bacteria, such as Fusobacterium and Raoultella, to enhance their virulence and colonization.
It has been associated with various diseases, like ulcerative colitis, colorectal cancer, and Crohn’s disease. However, the host defenses of Ruminococcus torques need to be better understood.Some studies have suggested that Ruminococcus torques may have some mechanisms to evade or modulate the host immune system, such as producing anti-inflammatory metabolites, altering the expression of host genes, and influencing the composition and function of other gut microbes. For example, Ruminococcus torques can produce butyrate, a short-chain fatty acid that has anti-inflammatory and immunomodulatory effects on the intestinal epithelium and immune cells. Ruminococcus torques can also affect the expression of host genes involved in inflammation, cell proliferation, and apoptosis, such as NF-κB, TNF-α, IL-6, IL-10, and Bcl-2. Additionally, Ruminococcus torques can interact with other gut bacteria, such as Faecalibacterium prausnitzii, Bacteroides caccae, and Bacteroides uniformis, and influence their abundance and function.
However, these mechanisms are not sufficient to protect Ruminococcus torques from the host defenses, and the bacteria may also face challenges from the host’s innate and adaptive immunity, such as mucosal barrier, antimicrobial peptides, phagocytes, and antibodies. Therefore, Ruminococcus torques may have to balance between survival and adaptation in the gut environment, and its role in health and disease may depend on the host’s genetic and environmental factors, as well as the interactions with other gut microbes. More research is needed to elucidate the host defenses of Ruminococcus torques and its implications for human health.
Ruminococcus torques is a type of bacteria that lives in the human gut. It belongs to the phylum Firmicutes and the order Clostridiales. It has been associated with various health conditions, like inflammatory bowel illness, colorectal cancer, and COVID-19. Here are some of the clinical manifestations of Ruminococcus torques:
A Mendelian randomization study found that the abundance of Ruminococcus torques was a risk factor for hospitalization and severe COVID-19. The study suggested that Ruminococcus torques might affect the immune response to SARS-CoV-2 infection by modulating the expression of genes involved in inflammation, viral replication, and cell adhesion.
A clinical investigation is being carried out right now to look into the effects of a Ruminococcus torques strain bacterium in healthy adults. The experiment is to assess the tolerability, safety, and immunomodulatory effects of the bacterium, as well as its impact on the gut microbiota composition and function.
A metagenomic biomarker based on the abundance of Ruminococcus torques and other gut bacteria was developed to predict the response to anti-PD-1 immunotherapy in patients with advanced melanoma. The biomarker showed a high accuracy in discriminating responders from non-responders. It revealed that Ruminococcus torques were positively connected, involving the infiltration of immune cells within the tumor microenvironment and immune checkpoint gene expression.
In the diagnosis of Ruminococcus torques infection, different methods can be used, such as:
Metagenomic analysis: This method involves sequencing the DNA of the microbial communities in the colonic tissue samples and identifying the species and genes present. A study found that Ruminococcus torques were a predictive biomarker for upper gastrointestinal tract involvement in patients with Crohn’s disease.
Culture-based methods: This method involves isolating and growing the bacteria from the fecal samples or biopsies and identifying them based on their morphology, biochemical characteristics, and molecular markers. A study described a new genus, Mediterraneibacter, and reclassified Ruminococcus torques as Mediterraneibacter torques based on culture and phylogenetic analysis.
Immunological methods: This method involves detecting the antibodies or antigens of the bacteria in the blood or stool samples using techniques such as enzyme-linked immunosorbent assay (ELISA) or immunofluorescence. A study found that Ruminococcus torques were involved in the modulation of enterochromaffin cells and serotonin levels in the caecum of patients with irritable bowel syndrome.
To prevent Ruminococcus torques in the gut, some possible strategies are:
Eating a diet rich in ellagic acid, a natural polyphenol found in fruits and vegetables such as pomegranates, raspberries, strawberries, and walnuts. Ellagic acid has been shown to improve antioxidant status, intestinal barrier function, and immune response in broilers under heat stress and to decrease the abundance of Ruminococcus torques in their cecum.
Taking probiotics that contain beneficial bacteria, such as Bifidobacterium, which can compete with Ruminococcus torques for nutrients and niches in the gut. Bifidobacterium has been found to be decreased in dementia patients who have Lewy bodies and may have protective effects on cognitive function and neuroinflammation.
Avoid excessive intake of dietary fiber, especially cellulose, which Ruminococcus torques and other cellulolytic bacteria in the gut can ferment. Excessive fermentation of fiber can lead to gas production, bloating, abdominal pain, and diarrhea. A moderate amount of fiber, however, is beneficial for gut health and regularity.
Frontiers | Development of a Novel Metagenomic Biomarker for Prediction of Upper Gastrointestinal Tract Involvement in Patients With Crohn’s Disease (frontiersin.org)
Mendelian Randomization Identifies Gut Ruminococcus Torques as a Causal Factor of Severe COVID-19 by Han Yan, Si Zhao, Han-Xue Huang, Pan Xie, Xin-He Cai, Yun-Dan Qu, Wei Zhang, Longbo Zhang, Xi Li :: SSRN
Ruminococcus torques are more abundant in the colonic tissue of patients with Crohn’s disease who have upper gastrointestinal tract involvement than those who do not. It may serve as a potential biomarker for predicting this complication.
Ruminococcus torques is also a risk factor for hospitalization and severe COVID-19, according to a Mendelian randomization study. It may affect the host’s immune response and inflammation through its metabolic pathways.
Ruminococcus torques are influenced by the spatial and temporal factors of the gut microbiota, such as the location of the sample, the age of the host, and the season of the year. It shows a higher relative abundance in the ileum than in the colon, in older hosts than in younger ones, and in winter than in summer.
Ruminococcus torques are involved in the pathogenesis of NAFLD, or non-alcoholic fatty liver disease, along with type 2 diabetes, a common comorbidity of diabetes4. It may alter the urine metabolites of the host, such as amino acids, fatty acids, and bile acids, which are related to glucose and lipid metabolism.
Kingdom: Bacteria
Phylum: Firmicutes
Class: Clostridia
Order: Clostridiales
Family: Ruminococcaceae
Genus: Ruminococcus
Species: R. torques
The structure of Ruminococcus torques are:
Ruminococcus torques is a Gram-positive bacterium, which means that it has a thick layer of peptidoglycan in its cell wall that retains the purple stain of the Gram staining method.
Ruminococcus torques is an anaerobic bacterium, which means that it does not require oxygen for its growth and metabolism. It can use alternative electron acceptors, such as nitrate, sulfate, or fumarate, to generate energy.
Ruminococcus torques is a rod-shaped bacterium, which means that it has a cylindrical or elongated morphology. It can form chains or clusters of cells, depending on the environmental conditions.
Ruminococcus torques is a cellulolytic bacterium, which means that it can degrade cellulose, a complex polysaccharide that forms the main component of plant cell walls. It produces enzymes, such as endoglucanases, cellobiohydrolases, and beta-glucosidases, that break down cellulose into glucose and other sugars.
Ruminococcus torques is a symbiont of the gut ecosystem, which means that it lives in association with other organisms, such as humans and other mammals, and provides them with some benefits, such as short-chain fatty acids, vitamins, and amino acids. It also influences the immune system, the intestinal barrier, and the brain function of the host.
The antigenic types of Ruminococcus torques are not well-studied. Still, some studies have suggested that it may have different strains based on the presence or absence of particular genes or metabolic pathways.
For example, a study by Togo et al. found that Ruminococcus torques ATCC 27756 had a gene cluster for the biosynthesis of exopolysaccharides, which may affect its antigenicity and adhesion to the intestinal mucosa. Another study by Ze et al. found that Ruminococcus torques were one of the dominant species involved in the human colon’s breakdown of resistant starch and that it had different metabolic profiles depending on the type of starch substrate.
It has been associated with various diseases, such as inflammatory bowel disease (IBD), irritable bowel syndrome (IBS), and type 2 diabetes mellitus (T2DM) with non-alcoholic fatty liver disease (NAFLD). The pathogenesis of Ruminococcus torques is not fully understood, but some possible mechanisms are:
Ruminococcus torques can degrade mucin, which is a protective layer of the intestinal epithelium, and increase the permeability of the gut barrier. It may allow the translocation of bacterial products and antigens into the bloodstream, triggering inflammation and immune responses.
Ruminococcus torques can produce short-chain fatty acids (SCFAs), such as the nutrients butyrate, propionate, and acetate are necessary for preserving intestinal homeostasis and adjusting host metabolism. However, excessive or imbalanced SCFAs may also have adverse effects, such as altering the gut pH, affecting intestinal motility, and influencing insulin sensitivity.
Ruminococcus torques can interact with other gut microbes and influence their composition and function. For example, Ruminococcus torques may compete with beneficial bacteria, such as Bifidobacterium and Lactobacillus, for nutrients and niches or cooperate with pathogenic bacteria, such as Fusobacterium and Raoultella, to enhance their virulence and colonization.
It has been associated with various diseases, like ulcerative colitis, colorectal cancer, and Crohn’s disease. However, the host defenses of Ruminococcus torques need to be better understood.Some studies have suggested that Ruminococcus torques may have some mechanisms to evade or modulate the host immune system, such as producing anti-inflammatory metabolites, altering the expression of host genes, and influencing the composition and function of other gut microbes. For example, Ruminococcus torques can produce butyrate, a short-chain fatty acid that has anti-inflammatory and immunomodulatory effects on the intestinal epithelium and immune cells. Ruminococcus torques can also affect the expression of host genes involved in inflammation, cell proliferation, and apoptosis, such as NF-κB, TNF-α, IL-6, IL-10, and Bcl-2. Additionally, Ruminococcus torques can interact with other gut bacteria, such as Faecalibacterium prausnitzii, Bacteroides caccae, and Bacteroides uniformis, and influence their abundance and function.
However, these mechanisms are not sufficient to protect Ruminococcus torques from the host defenses, and the bacteria may also face challenges from the host’s innate and adaptive immunity, such as mucosal barrier, antimicrobial peptides, phagocytes, and antibodies. Therefore, Ruminococcus torques may have to balance between survival and adaptation in the gut environment, and its role in health and disease may depend on the host’s genetic and environmental factors, as well as the interactions with other gut microbes. More research is needed to elucidate the host defenses of Ruminococcus torques and its implications for human health.
Ruminococcus torques is a type of bacteria that lives in the human gut. It belongs to the phylum Firmicutes and the order Clostridiales. It has been associated with various health conditions, like inflammatory bowel illness, colorectal cancer, and COVID-19. Here are some of the clinical manifestations of Ruminococcus torques:
A Mendelian randomization study found that the abundance of Ruminococcus torques was a risk factor for hospitalization and severe COVID-19. The study suggested that Ruminococcus torques might affect the immune response to SARS-CoV-2 infection by modulating the expression of genes involved in inflammation, viral replication, and cell adhesion.
A clinical investigation is being carried out right now to look into the effects of a Ruminococcus torques strain bacterium in healthy adults. The experiment is to assess the tolerability, safety, and immunomodulatory effects of the bacterium, as well as its impact on the gut microbiota composition and function.
A metagenomic biomarker based on the abundance of Ruminococcus torques and other gut bacteria was developed to predict the response to anti-PD-1 immunotherapy in patients with advanced melanoma. The biomarker showed a high accuracy in discriminating responders from non-responders. It revealed that Ruminococcus torques were positively connected, involving the infiltration of immune cells within the tumor microenvironment and immune checkpoint gene expression.
In the diagnosis of Ruminococcus torques infection, different methods can be used, such as:
Metagenomic analysis: This method involves sequencing the DNA of the microbial communities in the colonic tissue samples and identifying the species and genes present. A study found that Ruminococcus torques were a predictive biomarker for upper gastrointestinal tract involvement in patients with Crohn’s disease.
Culture-based methods: This method involves isolating and growing the bacteria from the fecal samples or biopsies and identifying them based on their morphology, biochemical characteristics, and molecular markers. A study described a new genus, Mediterraneibacter, and reclassified Ruminococcus torques as Mediterraneibacter torques based on culture and phylogenetic analysis.
Immunological methods: This method involves detecting the antibodies or antigens of the bacteria in the blood or stool samples using techniques such as enzyme-linked immunosorbent assay (ELISA) or immunofluorescence. A study found that Ruminococcus torques were involved in the modulation of enterochromaffin cells and serotonin levels in the caecum of patients with irritable bowel syndrome.
To prevent Ruminococcus torques in the gut, some possible strategies are:
Eating a diet rich in ellagic acid, a natural polyphenol found in fruits and vegetables such as pomegranates, raspberries, strawberries, and walnuts. Ellagic acid has been shown to improve antioxidant status, intestinal barrier function, and immune response in broilers under heat stress and to decrease the abundance of Ruminococcus torques in their cecum.
Taking probiotics that contain beneficial bacteria, such as Bifidobacterium, which can compete with Ruminococcus torques for nutrients and niches in the gut. Bifidobacterium has been found to be decreased in dementia patients who have Lewy bodies and may have protective effects on cognitive function and neuroinflammation.
Avoid excessive intake of dietary fiber, especially cellulose, which Ruminococcus torques and other cellulolytic bacteria in the gut can ferment. Excessive fermentation of fiber can lead to gas production, bloating, abdominal pain, and diarrhea. A moderate amount of fiber, however, is beneficial for gut health and regularity.
Frontiers | Development of a Novel Metagenomic Biomarker for Prediction of Upper Gastrointestinal Tract Involvement in Patients With Crohn’s Disease (frontiersin.org)
Mendelian Randomization Identifies Gut Ruminococcus Torques as a Causal Factor of Severe COVID-19 by Han Yan, Si Zhao, Han-Xue Huang, Pan Xie, Xin-He Cai, Yun-Dan Qu, Wei Zhang, Longbo Zhang, Xi Li :: SSRN
Ruminococcus torques are more abundant in the colonic tissue of patients with Crohn’s disease who have upper gastrointestinal tract involvement than those who do not. It may serve as a potential biomarker for predicting this complication.
Ruminococcus torques is also a risk factor for hospitalization and severe COVID-19, according to a Mendelian randomization study. It may affect the host’s immune response and inflammation through its metabolic pathways.
Ruminococcus torques are influenced by the spatial and temporal factors of the gut microbiota, such as the location of the sample, the age of the host, and the season of the year. It shows a higher relative abundance in the ileum than in the colon, in older hosts than in younger ones, and in winter than in summer.
Ruminococcus torques are involved in the pathogenesis of NAFLD, or non-alcoholic fatty liver disease, along with type 2 diabetes, a common comorbidity of diabetes4. It may alter the urine metabolites of the host, such as amino acids, fatty acids, and bile acids, which are related to glucose and lipid metabolism.
Kingdom: Bacteria
Phylum: Firmicutes
Class: Clostridia
Order: Clostridiales
Family: Ruminococcaceae
Genus: Ruminococcus
Species: R. torques
The structure of Ruminococcus torques are:
Ruminococcus torques is a Gram-positive bacterium, which means that it has a thick layer of peptidoglycan in its cell wall that retains the purple stain of the Gram staining method.
Ruminococcus torques is an anaerobic bacterium, which means that it does not require oxygen for its growth and metabolism. It can use alternative electron acceptors, such as nitrate, sulfate, or fumarate, to generate energy.
Ruminococcus torques is a rod-shaped bacterium, which means that it has a cylindrical or elongated morphology. It can form chains or clusters of cells, depending on the environmental conditions.
Ruminococcus torques is a cellulolytic bacterium, which means that it can degrade cellulose, a complex polysaccharide that forms the main component of plant cell walls. It produces enzymes, such as endoglucanases, cellobiohydrolases, and beta-glucosidases, that break down cellulose into glucose and other sugars.
Ruminococcus torques is a symbiont of the gut ecosystem, which means that it lives in association with other organisms, such as humans and other mammals, and provides them with some benefits, such as short-chain fatty acids, vitamins, and amino acids. It also influences the immune system, the intestinal barrier, and the brain function of the host.
The antigenic types of Ruminococcus torques are not well-studied. Still, some studies have suggested that it may have different strains based on the presence or absence of particular genes or metabolic pathways.
For example, a study by Togo et al. found that Ruminococcus torques ATCC 27756 had a gene cluster for the biosynthesis of exopolysaccharides, which may affect its antigenicity and adhesion to the intestinal mucosa. Another study by Ze et al. found that Ruminococcus torques were one of the dominant species involved in the human colon’s breakdown of resistant starch and that it had different metabolic profiles depending on the type of starch substrate.
It has been associated with various diseases, such as inflammatory bowel disease (IBD), irritable bowel syndrome (IBS), and type 2 diabetes mellitus (T2DM) with non-alcoholic fatty liver disease (NAFLD). The pathogenesis of Ruminococcus torques is not fully understood, but some possible mechanisms are:
Ruminococcus torques can degrade mucin, which is a protective layer of the intestinal epithelium, and increase the permeability of the gut barrier. It may allow the translocation of bacterial products and antigens into the bloodstream, triggering inflammation and immune responses.
Ruminococcus torques can produce short-chain fatty acids (SCFAs), such as the nutrients butyrate, propionate, and acetate are necessary for preserving intestinal homeostasis and adjusting host metabolism. However, excessive or imbalanced SCFAs may also have adverse effects, such as altering the gut pH, affecting intestinal motility, and influencing insulin sensitivity.
Ruminococcus torques can interact with other gut microbes and influence their composition and function. For example, Ruminococcus torques may compete with beneficial bacteria, such as Bifidobacterium and Lactobacillus, for nutrients and niches or cooperate with pathogenic bacteria, such as Fusobacterium and Raoultella, to enhance their virulence and colonization.
It has been associated with various diseases, like ulcerative colitis, colorectal cancer, and Crohn’s disease. However, the host defenses of Ruminococcus torques need to be better understood.Some studies have suggested that Ruminococcus torques may have some mechanisms to evade or modulate the host immune system, such as producing anti-inflammatory metabolites, altering the expression of host genes, and influencing the composition and function of other gut microbes. For example, Ruminococcus torques can produce butyrate, a short-chain fatty acid that has anti-inflammatory and immunomodulatory effects on the intestinal epithelium and immune cells. Ruminococcus torques can also affect the expression of host genes involved in inflammation, cell proliferation, and apoptosis, such as NF-κB, TNF-α, IL-6, IL-10, and Bcl-2. Additionally, Ruminococcus torques can interact with other gut bacteria, such as Faecalibacterium prausnitzii, Bacteroides caccae, and Bacteroides uniformis, and influence their abundance and function.
However, these mechanisms are not sufficient to protect Ruminococcus torques from the host defenses, and the bacteria may also face challenges from the host’s innate and adaptive immunity, such as mucosal barrier, antimicrobial peptides, phagocytes, and antibodies. Therefore, Ruminococcus torques may have to balance between survival and adaptation in the gut environment, and its role in health and disease may depend on the host’s genetic and environmental factors, as well as the interactions with other gut microbes. More research is needed to elucidate the host defenses of Ruminococcus torques and its implications for human health.
Ruminococcus torques is a type of bacteria that lives in the human gut. It belongs to the phylum Firmicutes and the order Clostridiales. It has been associated with various health conditions, like inflammatory bowel illness, colorectal cancer, and COVID-19. Here are some of the clinical manifestations of Ruminococcus torques:
A Mendelian randomization study found that the abundance of Ruminococcus torques was a risk factor for hospitalization and severe COVID-19. The study suggested that Ruminococcus torques might affect the immune response to SARS-CoV-2 infection by modulating the expression of genes involved in inflammation, viral replication, and cell adhesion.
A clinical investigation is being carried out right now to look into the effects of a Ruminococcus torques strain bacterium in healthy adults. The experiment is to assess the tolerability, safety, and immunomodulatory effects of the bacterium, as well as its impact on the gut microbiota composition and function.
A metagenomic biomarker based on the abundance of Ruminococcus torques and other gut bacteria was developed to predict the response to anti-PD-1 immunotherapy in patients with advanced melanoma. The biomarker showed a high accuracy in discriminating responders from non-responders. It revealed that Ruminococcus torques were positively connected, involving the infiltration of immune cells within the tumor microenvironment and immune checkpoint gene expression.
In the diagnosis of Ruminococcus torques infection, different methods can be used, such as:
Metagenomic analysis: This method involves sequencing the DNA of the microbial communities in the colonic tissue samples and identifying the species and genes present. A study found that Ruminococcus torques were a predictive biomarker for upper gastrointestinal tract involvement in patients with Crohn’s disease.
Culture-based methods: This method involves isolating and growing the bacteria from the fecal samples or biopsies and identifying them based on their morphology, biochemical characteristics, and molecular markers. A study described a new genus, Mediterraneibacter, and reclassified Ruminococcus torques as Mediterraneibacter torques based on culture and phylogenetic analysis.
Immunological methods: This method involves detecting the antibodies or antigens of the bacteria in the blood or stool samples using techniques such as enzyme-linked immunosorbent assay (ELISA) or immunofluorescence. A study found that Ruminococcus torques were involved in the modulation of enterochromaffin cells and serotonin levels in the caecum of patients with irritable bowel syndrome.
To prevent Ruminococcus torques in the gut, some possible strategies are:
Eating a diet rich in ellagic acid, a natural polyphenol found in fruits and vegetables such as pomegranates, raspberries, strawberries, and walnuts. Ellagic acid has been shown to improve antioxidant status, intestinal barrier function, and immune response in broilers under heat stress and to decrease the abundance of Ruminococcus torques in their cecum.
Taking probiotics that contain beneficial bacteria, such as Bifidobacterium, which can compete with Ruminococcus torques for nutrients and niches in the gut. Bifidobacterium has been found to be decreased in dementia patients who have Lewy bodies and may have protective effects on cognitive function and neuroinflammation.
Avoid excessive intake of dietary fiber, especially cellulose, which Ruminococcus torques and other cellulolytic bacteria in the gut can ferment. Excessive fermentation of fiber can lead to gas production, bloating, abdominal pain, and diarrhea. A moderate amount of fiber, however, is beneficial for gut health and regularity.
Frontiers | Development of a Novel Metagenomic Biomarker for Prediction of Upper Gastrointestinal Tract Involvement in Patients With Crohn’s Disease (frontiersin.org)
Mendelian Randomization Identifies Gut Ruminococcus Torques as a Causal Factor of Severe COVID-19 by Han Yan, Si Zhao, Han-Xue Huang, Pan Xie, Xin-He Cai, Yun-Dan Qu, Wei Zhang, Longbo Zhang, Xi Li :: SSRN
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