Anthropometric Measurements as Predictors of Low Birth Weight Among Tanzanian Neonates: A Hospital-Based Study
November 7, 2025
Background
Dermatosis is a broad term for skin diseases with diverse conditions and it is not specific to one disease.
It includes any irregularity or lesion on the skin. It occurs due to infections and irritation.
Types of Dermatoses are:
Genetic Dermatoses:
It occurs from genetic/inherited mutations.
E.g. Ichthyosis, epidermolysis bullosa
Infectious Dermatoses:
It is caused due to bacteria, viruses, fungi, or parasites.
E.g. Impetigo, herpes simplex, ringworm, scabies
Neoplastic Dermatoses:
It involves the growth of abnormal skin cells.
E.g. Melanoma, basal and squamous cell carcinoma
Inflammatory Dermatoses:
It is caused due to inflammation of the skin.
E.g. Eczema, psoriasis, dermatitis
The main symptoms of dermatoses are:
Redness, Swelling
Discoloration of skin
Nodules/lumps
Itching, Pain
Scaling/flaking skin
Epidemiology
Eczema is seen in 10% to 20% of cases in children and 1% to 3% in adults. While psoriasis affects 2% to 3% of the global population.
Acne vulgaris is common skin condition that affects 85% of individuals worldwide. Many people have cold sores or genital herpes from herpes virus.
Higher skin cancer cases are observed in sun-exposed regions such as Australia, New Zealand, North America, and Europe.
Skin diseases cause discomfort, distress, and social stigma that affects patient personal life.
Anatomy
Pathophysiology
Atopic dermatitis shows Th2 immune response. Filaggrin gene mutations give weak skin barrier to produce allergens and inflammation.
Immune response in psoriasis causes inflammation and keratinocyte proliferation. Keratinocyte dysfunction causes skin scaling and thickening.
Autoantibodies attack desmoglein in skin cells and causes adhesion loss and blisters. Inflammatory response worsens tissue damage.
Genetic mutations disrupt keratinization causes skin barrier dysfunction, increased water loss, and infection vulnerability.
Etiology
Causes of dermatosis are:
Genetic Factors as:
Eczema
Psoriasis
Hereditary Factors as:
Ichthyosis
Gene Mutations
Environmental Factors as:
Contact Dermatitis
Allergens and Irritants
Acne Vulgaris
Environmental Triggers as:
Diet
Skin Cancer
UV Radiation
Infectious Agents as:
Impetigo
Cellulitis
Herpes Simplex Virus
Dermatophytes
Candida
Genetics
Prognostic Factors
Severe diseases with large lesions need intensive treatment for improvement.
Early-onset skin conditions are chronic in nature thus difficult to treat.
E.g., eczema and psoriasis
Emotional stress can worsen skin conditions and treatment outcomes.
Clinical History
Dermatosis affects individuals of all age groups from infants to adults.
Physical Examination
Systematic Examination
Assessment of primary and secondary lesions
Nail Examination
Scalp Examination
Age group
Associated comorbidity
Associated activity
Acuity of presentation
For acute dermatoses:
Severe itching, pain, redness, and swelling
For chronic dermatoses:
Long-lasting, recurrent with remission and exacerbation
Differential Diagnoses
Erosions and Ulcers
Pustular Lesions
Nodules and Tumors
Erythematous Papules and Plaques
Vesicular and Bullous Lesions
Laboratory Studies
Imaging Studies
Procedures
Histologic Findings
Staging
Treatment Paradigm
Patients should identify and avoid triggers including allergens and irritants.
The goal is to control symptoms, promote healing and minimize recurrence to manage chronic condition.
The treatment phase is divided into topical, oral and systemic therapy.
Topical treatments include use of:
Corticosteroids are used in the treatment of eczema, psoriasis, and contact dermatitis.
Antibiotics and antifungal agents are used to treat impetigo and tinea.
Systemic treatments include use of:
Oral corticosteroids are indicated to treat severe eczema and lupus erythematosus.
Immunosuppressants are indicated to treat psoriasis and atopic dermatitis.
by Stage
by Modality
Chemotherapy
Radiation Therapy
Surgical Interventions
Hormone Therapy
Immunotherapy
Hyperthermia
Photodynamic Therapy
Stem Cell Transplant
Targeted Therapy
Palliative Care
use-of-non-pharmacological-approach-for-dermatosis
Patients should identify and avoid triggers including allergens and irritants.
The goal is to control symptoms, promote healing and minimize recurrence to manage chronic condition.
The treatment phase is divided into topical, oral and systemic therapy.
Topical treatments include use of:
Corticosteroids are used in the treatment of eczema, psoriasis, and contact dermatitis.
Antibiotics and antifungal agents are used to treat impetigo and tinea.
Systemic treatments include use of:
Oral corticosteroids are indicated to treat severe eczema and lupus erythematosus.
Immunosuppressants are indicated to treat psoriasis and atopic dermatitis.
Use of Topical Retinoids
It decreases cohesiveness of keratinocytes in sebaceous follicles for easy removal of acne.
Use of Topical Antifungals
It binds with phospholipids to change the permeability of fungal cell membrane.
Use of Topical Antibiotics
It binds with the 50S ribosomal subunits of susceptible bacteria to prevent elongation of peptide chains.
Use of Oral Corticosteroids
It controls the rate of protein synthesis to suppress migration of polymorphonuclear leukocytes and stabilizes lysosomes.
use-of-intervention-with-a-procedure-in-treating-dermatosis
Cryotherapy is used to treat benign skin lesions, warts, and precancerous lesions.
Phototherapy is indicated to treat psoriasis, eczema, and vitiligo.
Chemical peels and microneedling techniques are popular due to their effectiveness.
use-of-phases-in-managing-dermatosis
In the initial diagnosis phase, evaluation of medical history, physical examination and diagnostic test to confirm diagnosis.
Pharmacologic therapy is effective in the treatment phase as it includes use of topical corticosteroids, antifungals, antibiotics and therapies.
The regular follow-up visits with the dermatologist are schedule to check the improvement of patients along with treatment response.
Medication
Indiated for Dermatosis
Ointment/cream/lotion: Apply sparingly to affected area 2-3 times a day, discontinue therapy if control achieved and if no improvement is seen reassess diagnosis within 4 weeks.
Gel or foam: Apply two times a day, discontinue use when control is achieved.
Apply topically into the affected area of skin daily or every 12 hours
betamethasone + salicylic acidÂ
Lotion: Apply a thin coating to the affected scalp region twice daily.
Discontinue usage and evaluate the patient if symptoms disappear after a week.
Ointment: Apply a thin coating to the affected region twice a day.
Discontinue usage and evaluate the patient if symptoms disappear after a week.
Indicated for Dermatosis
For above 3 years age, apply foam or gel sparingly to affected area 2-3 times a day, discontinue therapy if control achieved.
Apply topically into the affected area of skin daily or every 12 hours
betamethasone + salicylic acidÂ
Lotion: Apply a thin coating to the affected scalp region twice daily.
Discontinue usage and evaluate the patient if symptoms disappear after a week.
Ointment: Apply a thin coating to the affected region twice a day.
Discontinue usage and evaluate the patient if symptoms disappear after a week.
Future Trends
Dermatosis is a broad term for skin diseases with diverse conditions and it is not specific to one disease.
It includes any irregularity or lesion on the skin. It occurs due to infections and irritation.
Types of Dermatoses are:
Genetic Dermatoses:
It occurs from genetic/inherited mutations.
E.g. Ichthyosis, epidermolysis bullosa
Infectious Dermatoses:
It is caused due to bacteria, viruses, fungi, or parasites.
E.g. Impetigo, herpes simplex, ringworm, scabies
Neoplastic Dermatoses:
It involves the growth of abnormal skin cells.
E.g. Melanoma, basal and squamous cell carcinoma
Inflammatory Dermatoses:
It is caused due to inflammation of the skin.
E.g. Eczema, psoriasis, dermatitis
The main symptoms of dermatoses are:
Redness, Swelling
Discoloration of skin
Nodules/lumps
Itching, Pain
Scaling/flaking skin
Eczema is seen in 10% to 20% of cases in children and 1% to 3% in adults. While psoriasis affects 2% to 3% of the global population.
Acne vulgaris is common skin condition that affects 85% of individuals worldwide. Many people have cold sores or genital herpes from herpes virus.
Higher skin cancer cases are observed in sun-exposed regions such as Australia, New Zealand, North America, and Europe.
Skin diseases cause discomfort, distress, and social stigma that affects patient personal life.
Atopic dermatitis shows Th2 immune response. Filaggrin gene mutations give weak skin barrier to produce allergens and inflammation.
Immune response in psoriasis causes inflammation and keratinocyte proliferation. Keratinocyte dysfunction causes skin scaling and thickening.
Autoantibodies attack desmoglein in skin cells and causes adhesion loss and blisters. Inflammatory response worsens tissue damage.
Genetic mutations disrupt keratinization causes skin barrier dysfunction, increased water loss, and infection vulnerability.
Causes of dermatosis are:
Genetic Factors as:
Eczema
Psoriasis
Hereditary Factors as:
Ichthyosis
Gene Mutations
Environmental Factors as:
Contact Dermatitis
Allergens and Irritants
Acne Vulgaris
Environmental Triggers as:
Diet
Skin Cancer
UV Radiation
Infectious Agents as:
Impetigo
Cellulitis
Herpes Simplex Virus
Dermatophytes
Candida
Severe diseases with large lesions need intensive treatment for improvement.
Early-onset skin conditions are chronic in nature thus difficult to treat.
E.g., eczema and psoriasis
Emotional stress can worsen skin conditions and treatment outcomes.
Dermatosis affects individuals of all age groups from infants to adults.
Systematic Examination
Assessment of primary and secondary lesions
Nail Examination
Scalp Examination
For acute dermatoses:
Severe itching, pain, redness, and swelling
For chronic dermatoses:
Long-lasting, recurrent with remission and exacerbation
Erosions and Ulcers
Pustular Lesions
Nodules and Tumors
Erythematous Papules and Plaques
Vesicular and Bullous Lesions
Patients should identify and avoid triggers including allergens and irritants.
The goal is to control symptoms, promote healing and minimize recurrence to manage chronic condition.
The treatment phase is divided into topical, oral and systemic therapy.
Topical treatments include use of:
Corticosteroids are used in the treatment of eczema, psoriasis, and contact dermatitis.
Antibiotics and antifungal agents are used to treat impetigo and tinea.
Systemic treatments include use of:
Oral corticosteroids are indicated to treat severe eczema and lupus erythematosus.
Immunosuppressants are indicated to treat psoriasis and atopic dermatitis.
Dermatology, General
Patients should identify and avoid triggers including allergens and irritants.
The goal is to control symptoms, promote healing and minimize recurrence to manage chronic condition.
The treatment phase is divided into topical, oral and systemic therapy.
Topical treatments include use of:
Corticosteroids are used in the treatment of eczema, psoriasis, and contact dermatitis.
Antibiotics and antifungal agents are used to treat impetigo and tinea.
Systemic treatments include use of:
Oral corticosteroids are indicated to treat severe eczema and lupus erythematosus.
Immunosuppressants are indicated to treat psoriasis and atopic dermatitis.
Dermatology, General
It decreases cohesiveness of keratinocytes in sebaceous follicles for easy removal of acne.
Dermatology, General
It binds with phospholipids to change the permeability of fungal cell membrane.
Dermatology, General
It binds with the 50S ribosomal subunits of susceptible bacteria to prevent elongation of peptide chains.
Dermatology, General
It controls the rate of protein synthesis to suppress migration of polymorphonuclear leukocytes and stabilizes lysosomes.
Dermatology, General
Cryotherapy is used to treat benign skin lesions, warts, and precancerous lesions.
Phototherapy is indicated to treat psoriasis, eczema, and vitiligo.
Chemical peels and microneedling techniques are popular due to their effectiveness.
Dermatology, General
In the initial diagnosis phase, evaluation of medical history, physical examination and diagnostic test to confirm diagnosis.
Pharmacologic therapy is effective in the treatment phase as it includes use of topical corticosteroids, antifungals, antibiotics and therapies.
The regular follow-up visits with the dermatologist are schedule to check the improvement of patients along with treatment response.
Dermatosis is a broad term for skin diseases with diverse conditions and it is not specific to one disease.
It includes any irregularity or lesion on the skin. It occurs due to infections and irritation.
Types of Dermatoses are:
Genetic Dermatoses:
It occurs from genetic/inherited mutations.
E.g. Ichthyosis, epidermolysis bullosa
Infectious Dermatoses:
It is caused due to bacteria, viruses, fungi, or parasites.
E.g. Impetigo, herpes simplex, ringworm, scabies
Neoplastic Dermatoses:
It involves the growth of abnormal skin cells.
E.g. Melanoma, basal and squamous cell carcinoma
Inflammatory Dermatoses:
It is caused due to inflammation of the skin.
E.g. Eczema, psoriasis, dermatitis
The main symptoms of dermatoses are:
Redness, Swelling
Discoloration of skin
Nodules/lumps
Itching, Pain
Scaling/flaking skin
Eczema is seen in 10% to 20% of cases in children and 1% to 3% in adults. While psoriasis affects 2% to 3% of the global population.
Acne vulgaris is common skin condition that affects 85% of individuals worldwide. Many people have cold sores or genital herpes from herpes virus.
Higher skin cancer cases are observed in sun-exposed regions such as Australia, New Zealand, North America, and Europe.
Skin diseases cause discomfort, distress, and social stigma that affects patient personal life.
Atopic dermatitis shows Th2 immune response. Filaggrin gene mutations give weak skin barrier to produce allergens and inflammation.
Immune response in psoriasis causes inflammation and keratinocyte proliferation. Keratinocyte dysfunction causes skin scaling and thickening.
Autoantibodies attack desmoglein in skin cells and causes adhesion loss and blisters. Inflammatory response worsens tissue damage.
Genetic mutations disrupt keratinization causes skin barrier dysfunction, increased water loss, and infection vulnerability.
Causes of dermatosis are:
Genetic Factors as:
Eczema
Psoriasis
Hereditary Factors as:
Ichthyosis
Gene Mutations
Environmental Factors as:
Contact Dermatitis
Allergens and Irritants
Acne Vulgaris
Environmental Triggers as:
Diet
Skin Cancer
UV Radiation
Infectious Agents as:
Impetigo
Cellulitis
Herpes Simplex Virus
Dermatophytes
Candida
Severe diseases with large lesions need intensive treatment for improvement.
Early-onset skin conditions are chronic in nature thus difficult to treat.
E.g., eczema and psoriasis
Emotional stress can worsen skin conditions and treatment outcomes.
Dermatosis affects individuals of all age groups from infants to adults.
Systematic Examination
Assessment of primary and secondary lesions
Nail Examination
Scalp Examination
For acute dermatoses:
Severe itching, pain, redness, and swelling
For chronic dermatoses:
Long-lasting, recurrent with remission and exacerbation
Erosions and Ulcers
Pustular Lesions
Nodules and Tumors
Erythematous Papules and Plaques
Vesicular and Bullous Lesions
Patients should identify and avoid triggers including allergens and irritants.
The goal is to control symptoms, promote healing and minimize recurrence to manage chronic condition.
The treatment phase is divided into topical, oral and systemic therapy.
Topical treatments include use of:
Corticosteroids are used in the treatment of eczema, psoriasis, and contact dermatitis.
Antibiotics and antifungal agents are used to treat impetigo and tinea.
Systemic treatments include use of:
Oral corticosteroids are indicated to treat severe eczema and lupus erythematosus.
Immunosuppressants are indicated to treat psoriasis and atopic dermatitis.
Dermatology, General
Patients should identify and avoid triggers including allergens and irritants.
The goal is to control symptoms, promote healing and minimize recurrence to manage chronic condition.
The treatment phase is divided into topical, oral and systemic therapy.
Topical treatments include use of:
Corticosteroids are used in the treatment of eczema, psoriasis, and contact dermatitis.
Antibiotics and antifungal agents are used to treat impetigo and tinea.
Systemic treatments include use of:
Oral corticosteroids are indicated to treat severe eczema and lupus erythematosus.
Immunosuppressants are indicated to treat psoriasis and atopic dermatitis.
Dermatology, General
It decreases cohesiveness of keratinocytes in sebaceous follicles for easy removal of acne.
Dermatology, General
It binds with phospholipids to change the permeability of fungal cell membrane.
Dermatology, General
It binds with the 50S ribosomal subunits of susceptible bacteria to prevent elongation of peptide chains.
Dermatology, General
It controls the rate of protein synthesis to suppress migration of polymorphonuclear leukocytes and stabilizes lysosomes.
Dermatology, General
Cryotherapy is used to treat benign skin lesions, warts, and precancerous lesions.
Phototherapy is indicated to treat psoriasis, eczema, and vitiligo.
Chemical peels and microneedling techniques are popular due to their effectiveness.
Dermatology, General
In the initial diagnosis phase, evaluation of medical history, physical examination and diagnostic test to confirm diagnosis.
Pharmacologic therapy is effective in the treatment phase as it includes use of topical corticosteroids, antifungals, antibiotics and therapies.
The regular follow-up visits with the dermatologist are schedule to check the improvement of patients along with treatment response.

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